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过氧化物歧化酶对CVB3m病毒所致的小鼠心肌损伤的保护作用
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缺血预适应心肌保护机制研究
预先反复、短暂缺血可以减轻后续长期缺血所造成组织损伤的现象被称为缺血预适应(ischemic preconditioning,IPC),包括IPC后即刻出现并持续1~3小时的早期保护和IPC后24小时再度出现并持续72小时的延迟保护作用两个时相.本项目主要研究老年、病变心脏IPC保护特征与规律,研究心脏缺血预适应延迟保护的细胞核反应机制,并探讨具有临床应用前景的药物预适应的保护机制,为临床上缺血性心脏病的防治提供新的思路.
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综合治疗脑卒中后肩手综合征的疗效
Background:The patients after hemodialysis may be caused malnutrition for decreased nutrition intake, nutrition loss during the hemodialysis, consumption caused by the disease, metabolic disorder and endocrine disorder etc. Malnutrition may caused lowered immunological function, long terms of anemia and hypoproteinemia may cause malnutrition of the cardiac muscle, lower cardiac function and risk heart failure. Malnutrition may also harm appetite and cause the malnutrition worse. Malnutrition caused insufficient vigor in the patients and affect their daily living abilities directly. So to regulate the nutrition level scientifically is important for ameliorating nutrition condition of the patients and to increase their daily living abilities.
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1,6-二磷酸果糖(FDP)对缺血心肌的保护及其作用机制
自从1980年Markov等首次观察了外源性1,6-二磷酸果糖(FDP)对缺血性心肌的保护作用后,FDP在心血管系统的作用已经从基础研究到临床应用全面展开.FDP已为国内外许多研究证明具有抗氧自由基的作用,能保护氧自由基对缺血再灌心肌的损伤,也有实验证明FDP抑制氧自由基生成,但是产生心肌缺血再灌损伤病变的核心是由于心肌细胞内游离钙离子(Ca2+)的积聚,所谓"钙超负荷".