发表一篇学和医学成像类SCI论文
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Abstract:
BACKGROUND:Evidence of sympathetic and renin-angiotensin-aldosterone system activation provided a rationale for neurohormonal antagonism in heart failure with reduced ejection fraction (HFrEF), while no data are available in patients with milder degree of systolic dysfunction. We aimed to investigate neurohormonal function in HF with preserved and mid-range EF (HFpEF/HFmrEF). METHODS:Three cohorts (n = 189/each) of stable HFpEF, HFmrEF and HFrEF patients were selected (median age 70, 67 and 67 years; male 56%, 73% and 74%, respectively). Patients received a baseline clinical assessment including plasma renin activity (PRA), aldosterone, catecholamines, and N-terminal fraction of pro-B-type natriuretic peptide (NT-proBNP) assays, and were followed-up for all-cause death. RESULTS:Neuroendocrine profile was similar between HFpEF and HFmrEF, while all neurohormones except epinephrine were higher in HFrEF than in HFmrEF (NT-proBNP 2332 ng/L, IQR 995-5666 vs 575 ng/L, 205-1714; PRA 1.7 ng/mL/h, 0.4-5.6 vs 0.6 ng/mL/h, 0.2-2.6; aldosterone 153 ng/L, 85-246 vs 113 ng/L, 72-177; norepinephrine 517 ng/L, 343-844 vs 430 ng/L, 259-624; all p < 0.001, epinephrine 31 ng/L, 10-63 vs 25 ng/L, 10-44; p = 0.319). These findings were unrelated to treatment heterogeneity. Ten percent of HFpEF patients had elevated PRA, aldosterone and norepinephrine vs. 8% in HFmrEF and 21% in HFrEF. During a 5-year follow-up, survival decreased with the number of neurohormones elevated (HFpEF: log-rank 7.8, p = 0.048; HFmrEF: log-rank 11.8, p = 0.008; HFrEF: log-rank 8.1, p = 0.044). CONCLUSIONS:Neurohormonal activation is present only in a subset of patients with HFpEF and HFmrEF, and may hold clinical significance. Neurohormonal antagonism may be useful in selected HFpEF/HFmrEF population.
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最新影响因子:4.039 | 期刊ISSN:0167-5273 | CiteScore:1.76 |
出版周期:Semimonthly | 是否OA:YES | 出版年份:1981 |
期刊官方网址:http://www.elsevier.com/wps/find/journaldescription.cws_home/506041/description#description
期刊投稿地址:http://ees.elsevier.com/ijc/
自引率:8.80% | 研究方向:医学-心血管系统 |
出版地区:IRELAND |
SCI期刊coverage:Science Citation Index Expanded(科学引文索引扩展)
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The International Journal of Cardiology is devoted to cardiology in the broadest sense. Both basic research and clinical papers can be submitted. The journal serves the interest of both practicing clinicians and research workers.Editorials, Brief Reports and Review Articles covering recent developments are included. Controversial techniques, issues on health policy and social medicine are discussed and serve as useful tools for encouraging debate.International Journal of Cardiology has no page charges.A reduced personal subscription rate is available; please apply to the Publisher for more information.
《国际心脏病学杂志》在最广泛的意义上致力于心脏病学。基础研究和临床论文都可以提交。该杂志为临床医师和研究工作者提供了服务,包括社论、简要报告和评论文章。讨论有争议的技术、健康政策和社会医学问题,并作为鼓励辩论的有用工具。国际心脏病杂志不收取页面费用。可降低个人订阅率;请向出版商申请了解更多信息。
大类(学科) | 小类(学科) | 学科排名 |
医学 |
CARDIAC & CARDIOVASCULAR SYSTEMS (心脏和心血管系统) 3区 |
41/128 |
年度总发文量 | 年度论文发表量 | 年度综述发表量 |
1199 | 1085 | 114 |
引文计数(2018)
文献(2015-2017)
11795次引用
6683篇文献
序号 | 类别 | 排名 | 百分位 |
1 |
大类(学科):Medicine
小类(学科):Cardiology and Cardiovascular Medicine
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#113/327
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研究方向:医学 儿科
审稿时间: 6个月内
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研究方向:抗凝 抗栓 药物基因组学
审稿时间: 2个月内 接受率: 比较困难(25%命中)
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审稿时间: 2个月内 接受率: 比较困难(25%命中)
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审稿时间: 3个月内
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审稿时间: 2个月内 接受率: 比较困难(25%命中)
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审稿时间: 3个月内 接受率: 中等(50%命中)
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影响因子:5.315
ISSN:0148-396X
研究方向:医学-临床神经学
影响因子:1.725
ISSN:0028-3770
研究方向:医学-临床神经学
影响因子:2.21
ISSN:1878-8750
研究方向:CLINICAL NEUROLOGY-SURGERY
影响因子:4.332
ISSN:1092-0684
研究方向:CLINICAL NEUROLOGY-SURGERY
影响因子:2.816
ISSN:0001-6268
研究方向:医学-临床神经学
影响因子:2.8
ISSN:0344-5607
研究方向:医学-临床神经学
影响因子:0.972
ISSN:1019-5149
研究方向:医学-临床神经学
影响因子:1.532
ISSN:0256-7040
研究方向:医学-临床神经学
影响因子:1.165
ISSN:1016-2291
研究方向:医学-临床神经学
发表一篇学和医学成像类SCI论文
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